A treatise on the nervous diseases of children, for physicians and students . in the cord. At eachlevel of the spinal cord a connec-tion is established between thepyramidal tracts in the lateral andanterior columns and the graymatter in the anterior horns ofthe spinal cord. The ganglioncells in this gray matter connectwith the anterior nerve - roots ;these pass through the spinal cov-erings, after joining with the sen-sory fibres which come from theposterior portion of the cord, andtogether with these and with sim-iliar fibres from other segments ofthe cord, form the various nerve-plexuses fro
A treatise on the nervous diseases of children, for physicians and students . in the cord. At eachlevel of the spinal cord a connec-tion is established between thepyramidal tracts in the lateral andanterior columns and the graymatter in the anterior horns ofthe spinal cord. The ganglioncells in this gray matter connectwith the anterior nerve - roots ;these pass through the spinal cov-erings, after joining with the sen-sory fibres which come from theposterior portion of the cord, andtogether with these and with sim-iliar fibres from other segments ofthe cord, form the various nerve-plexuses from which the nerve-trunk issues. Every portion of this motor tract, from the cortex ofthe brain to the peripheral muscles, has been thoroughlvstudied and its exact location firmly established. We candivide the motor path for any one muscle of the bodvinto two distinct parts. This is not an anatomical ca-price but a division of the utmost importance from a purelyclinical point of view. The first division extends fromthe cortex of the brain through the lateral columns of the. Fig. 126. — Diagram representingMotor Innervation of a Muscle,and the Two Divisions of theMotor Tract. (After a figurefrom Edinger.) 454 THE NERVOUS DISEASES OF CHILDREN. cord to the ganglion cells of the anterior horns; the sec-ond division from the same ganglion cells to the first attempt in the way of localization of a lesion caus-ing a paralysis should be to determine whether the symp-toms point to a lesion in Division I. or in Division II. ofthe motor tract. (Fig. 126.) A lesion anywhere in Part rise to spastic paralysis (without atrophy) and to in-creased reflexes, but the electrical reactions of the para-lyzed parts remain entirely normal. A lesion anywhere inDivision II. causes an atrophic and flaccid paralysis; the re-flexes instead of being increased are diminished, the electri-cal reactions, far from being normal, are distinctly altered.(For an explanation of these facts see
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Keywords: ., bookcentury1800, bookdecade1890, booksubjectnervous, bookyear1895
