Nervous and mental diseases . Left. Right, Fig. 30.—Blindness in both temporal fields in a case of acromegalia due to injury of the chiasm bypituitary enlargement. The nasal fields are also contracted. lateral halves of the retinas on the same side of each eyeball. Anylesion that interrupts the visual pathway back of the chiasm, or destroysthe visual centers in the cuneus, produces lateral homonymous Left. Right- Fig. 31.—Homonymous lateral hemianopsia from an injury to the right occipital apex. The right lateral fields also contracted. The cortical half-vision centers of the cu


Nervous and mental diseases . Left. Right, Fig. 30.—Blindness in both temporal fields in a case of acromegalia due to injury of the chiasm bypituitary enlargement. The nasal fields are also contracted. lateral halves of the retinas on the same side of each eyeball. Anylesion that interrupts the visual pathway back of the chiasm, or destroysthe visual centers in the cuneus, produces lateral homonymous Left. Right- Fig. 31.—Homonymous lateral hemianopsia from an injury to the right occipital apex. The right lateral fields also contracted. The cortical half-vision centers of the cuneus are in turn brought byconnecting fibers into relation with higher centers for visual memories,probably situated in and about the angular gyri of the parietal these higher visual centers both eyes are represented in each hemi- 104 DISEASES OF THE CRANIAL NERVES. sphere. The parietal centers probably freely communicate through thecallosal crossway. Injury to the left parietal region in right-handedpersons produces loss of visual word-memories, or word-blindness, butdoes not necessarily cause hemianopsia. The fibers which supply the macula lutea of the retina occupy at theapex of the orbit the central portion of the optic nerve in close proxim-ity to the central artery and vein. They then become superficial on theouter side of the nerve and proceed in this position to the back of theeye. Aneury


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