. The elements of pathological histology with special reference to practical methods . Fig. 99.—The Thrombus in Fig. 97 more highly magnified (x 240). o, Thrombus,composed of blood-platelets and leucocytes; 6, Proliferated stellate and spindle-shapedconnective-tissue cells of the valve. Myocarditis is set up either by the extension of an endocarditisto the myocardium, or as a consequence of embolism; that is, whenthe specific excitants of some mycotic inflammation, existing inanother part of the body, succeed in reaching the arteries of the the latter case foci form which are numerous
. The elements of pathological histology with special reference to practical methods . Fig. 99.—The Thrombus in Fig. 97 more highly magnified (x 240). o, Thrombus,composed of blood-platelets and leucocytes; 6, Proliferated stellate and spindle-shapedconnective-tissue cells of the valve. Myocarditis is set up either by the extension of an endocarditisto the myocardium, or as a consequence of embolism; that is, whenthe specific excitants of some mycotic inflammation, existing inanother part of the body, succeed in reaching the arteries of the the latter case foci form which are numerous, though mostly very ACUTE PERICARDITIS 199 minute, whilst in the former the inflammation may also attain aconsiderable extent. The embolic foci (Fig. 100) consist of bacteria. Fio. 100.—Metastatic Myocarditis in Pyemia, x 545; the cocci drawn in undera power of x 180 times. (Weigerts modification of Grams method.) a, Staphylococci;6, Necrotic tissue ; c, Round cells ; d, Necrotic primitive muscular bundles, with small-celled infiltration of the interstitial connective tissue; e, normal primitive muscularbundles. ia) and necrotic (often waxy-degenerated) muscle fibres {d), togetherwith a small-celled infiltration (c), which latter, if due to the action ofpus bacteria, soon assumes a purulent character, so that the foci growinto abscesses of larger or smaller size. The latter may advance untilthey lead to rupture of the heart, or else may heal up by cicatrisation,with or without calcification. Acute pericarditis is in the majority of cases of secondary origin,being due either to the extension of inflammation to the pericardiumfrom a neighbouring part, most frequently the pleura, peritoneum,or heart itself, or to the entrance into the circulation of the spec
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Keywords: ., bookcentury1800, bookdecade1890, booksubjectpatholo, bookyear1895
