. Cyclopædia of obstetrics and gynecology . true of foetal placentitis. Cauwen-berghe regards it as doubtful; Duchamp admits its existence, with thereservation that, although the suppurative form is doubtful, the chronicor sclerotic form is real. It shows itself in fibrous degeneration of thevilli. The cases of so-called abscess of the placenta, numbering ten inscientific literature, are questionable, for not one of the authors remem- DISEASES OF THE OVU>r. 225 bered to analyze the pus, and it is more than probable that it was notgenuine pus but what Eobin has called pseudo-fibrinous pus. 2
. Cyclopædia of obstetrics and gynecology . true of foetal placentitis. Cauwen-berghe regards it as doubtful; Duchamp admits its existence, with thereservation that, although the suppurative form is doubtful, the chronicor sclerotic form is real. It shows itself in fibrous degeneration of thevilli. The cases of so-called abscess of the placenta, numbering ten inscientific literature, are questionable, for not one of the authors remem- DISEASES OF THE OVU>r. 225 bered to analyze the pus, and it is more than probable that it was notgenuine pus but what Eobin has called pseudo-fibrinous pus. 2. Changes in tJie Chorion. The maternal placenta is formed by the decidua serotina. The foetalplacenta is formed by the villi of the chorion which, having originallycovered the entire surface of the ovum, atrophy over the major part ofthe surface, while they ramify and develop, ad infinitum, at the pointcorresponding to the serotina, where they become imbedded and consti-tute the vascular mass known as the placenta. To study changes in the. Fig. 21.—Bloody Mole, with Extravasations and Blood-cysts on the Fcetal Surface.(After SpieQelberg.)—CS, Clots. KH, Blood Cysts. chorion amounts to studying the lesions of the placenta and the , these changes may relate to each of the placental elements, ,the vessels and the villi. Let us successively study these lesions. I. Atrophy of the Villi of the Chorion. This atrophy occurs, normally, in all the villi not destined to form theplacenta, , in all not in contact with the serotina, and these villi maypresent three different conditions: 1. They are vascular. 2. They are well-formed and hollow but non-vascular. 3. They are mal-formed, and this has prevented their atrophy is different in the vascular and in the non-vascular villi. Ch. Eobin has best described this atrophy: 1. If the villus is non-vas-cular, it is obliterated and undergoes fatty degeneration; 2. If the villusis vascular, the vessels
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