. The thyroid gland in health and disease. FiG. 23.—A further stage in the degenerative process. Section of thyroid froma goitrous rat fed on fsecal material. Note ceUular disintegration, complete absenceof acini or colloid, increase of the fibrous stroma. A large distended vessel is alsoseen. Appearance corresponds to that seen in figs. 9, 24, 51, with which Fig. 24.—Final stage in the degenerative process. Section of thyroid from acase of myxoedema. Note absence of vesicles and colloid, atrophy of parenchymaand great increase of fibrous tissue. From a specimen in the possessi
. The thyroid gland in health and disease. FiG. 23.—A further stage in the degenerative process. Section of thyroid froma goitrous rat fed on fsecal material. Note ceUular disintegration, complete absenceof acini or colloid, increase of the fibrous stroma. A large distended vessel is alsoseen. Appearance corresponds to that seen in figs. 9, 24, 51, with which Fig. 24.—Final stage in the degenerative process. Section of thyroid from acase of myxoedema. Note absence of vesicles and colloid, atrophy of parenchymaand great increase of fibrous tissue. From a specimen in the possession of the lateSir Victor Horsley, 74 PATHOLOGY It will be recognized that every grade of severity, aciitenessor chronicity of this continued excitation-process may occur, aswell as every grade of resultant thyroid defect, dependent nodoubt upon the nature and dose of the toxic excitant and thecoexistence of other influences which excite and ultimatelyimpair thyroidal activity. Ultimate recovery or ultimate celldeath will also to a great extent be influenced by the coexistenceof the other factors to which reference has been made in thepreceding sections. The changes in the foetal gland which give rise to cretinism,are essentiallly the same as those just described (figs. 25, 64),and are due to toxic agencies acting through the medium of thematernal blood. Thei
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