. The elements of pathological histology with special reference to practical methods . cnes(see p. 119). ?^7 >•---. • * %\MSi Fig. 103.—Secondary Vasculitis of a Small Vein of the Leg in Phlegmon dueto Streptococcus pyogenes. X 545 ; the cocci, however, drawn in under an amplificationof X 765. (Weigerts modification of Grams method.) «, Accumulation of leucocytes,in part polynuclear, in the lumen of the vein ; b, Streptococci. When the smaller blood-vessels (Fig. 103) are involved, thereoccurs at first an ever-increasing accumulation of leucocytes (a) intheir lumen, and, should this be furt
. The elements of pathological histology with special reference to practical methods . cnes(see p. 119). ?^7 >•---. • * %\MSi Fig. 103.—Secondary Vasculitis of a Small Vein of the Leg in Phlegmon dueto Streptococcus pyogenes. X 545 ; the cocci, however, drawn in under an amplificationof X 765. (Weigerts modification of Grams method.) «, Accumulation of leucocytes,in part polynuclear, in the lumen of the vein ; b, Streptococci. When the smaller blood-vessels (Fig. 103) are involved, thereoccurs at first an ever-increasing accumulation of leucocytes (a) intheir lumen, and, should this be further followed by deposition offibrin, a so-called white thromhus is formed (see p. 208), which again, 204 THE BLOOD-VESSELS should pyococci have gained entrance into the vessel, eventuallyundergoes purulent liquefaction. In addition to this, leucocytes willalso emigrate into the wall of the vessel and into the perivascularconnective tissue. Should, however, an inflammation due to pyococci extend to blood-vessels of larger size (Fig. 104), an active growth of the penetrating. Fio. 104. —Secondary Vasculitis of the Innominate Artery, with rupture of thevessel, due to Streptococcus ppogenes, starting from a tracheotomy wound in a diph-theritic child. X 440 ; the cocci drawn in under a power of x 980. (Weigerts modifica-tion of Grams method.) a, Necrotic and undermintd intiiiia ; b, Small-celled infiltrationof intima; c, Fissures and cavities between the necrotic portions of the wall of thevessel ; (/, Torn and separated necrotic portions of the wall: e, Cocci. bacteria (e) may ensue in the walls of the latter, the further con-sequences of which may be partly necrosis (a) and partly purulentinfiltration (b) of the coats of the vessel. As the powers of resistanceof the wall are presumably greatly reduced by this process, theremay occur, before the lumen of the vessel has yet become occludedby thrombus as already described, either an immediate rupture ofthe vessel, or, the ou
Size: 1500px × 1666px
Photo credit: © Reading Room 2020 / Alamy / Afripics
License: Licensed
Model Released: No
Keywords: ., bookcentury1800, bookdecade1890, booksubjectpatholo, bookyear1895
