A system of obstetrics . d not the facts, is at fault. The fibrouschange, consisting in the displacement of the normal structures byfibrous tissue, by the atrophy of the former either from the pressureexerted by the hypertrophied fibrous elements or by their subsequentcontraction, may originate either in the decidua serotina, the placentalvilli, or the intervillous spaces. When the disease affects the decidua serotina, it is associated withchronic inflammation of the remainder of the endometrium, and it would be better perhaps, withBraun, Schroeder, and Spiegel-berg, to regard this not as a di
A system of obstetrics . d not the facts, is at fault. The fibrouschange, consisting in the displacement of the normal structures byfibrous tissue, by the atrophy of the former either from the pressureexerted by the hypertrophied fibrous elements or by their subsequentcontraction, may originate either in the decidua serotina, the placentalvilli, or the intervillous spaces. When the disease affects the decidua serotina, it is associated withchronic inflammation of the remainder of the endometrium, and it would be better perhaps, withBraun, Schroeder, and Spiegel-berg, to regard this not as a dis-ease of the placenta, but as anendometritis. As the disease pro-gresses, however, the placenta be-comes secondarily involved, eitherby the encroachment of the hy-pertrophied decidua upon the in-tervillous spaces, and the conse-quent compression of the villi, orby the agglutination of the dif-ferent layers of the membrane oneto another, which may result inthe firm adherence of the placenta to the uterine wall. Hegar and. The Maternal Portion of the Placenta, showingelongated decidual cells, the first step ininterstitial placentitis. THE PLACENTA. 245 Maier, and many others since them, have described this disease asinterstitial The same microscopic appearance may be seen in an hypertrophieddecidua throughout its extent, and is not confined to the placental is, however, possible to find an endometrium in an advanced stageof hyperplastic inflammation, while the upper layer of the deciduaserotina remains unchanged, even although the placental site itself isimmensely thickened by new-formed connective tissue and enlargedblood-sinuses. In such a case, an example of which I have seen, theplacenta remains unaffected. The fibrous degeneration may have its seat in the placental villialone. The process that transforms a healthy villus containing blood-vessels into a bundle of connective tissue can well be studied in theextra-placental villi of the chorion, which no
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Keywords: ., bookcentury1800, bookdecade1880, booksubjectobstetrics, bookyear1
