Clinical electrocardiography . Fig. 84.—Right bundle branch Fig. 85.—Left bundle branch block. subendocardial lesions were produced without the developmentof Q R S changes. Abnormal complexes were obtained, however, 106 CLINICAL ELECTROCARDIOGRAPHY by dividing the bundle branches, and the complexes becamedefinitely bizarre when ventricular dilatation supervened. Smithconcluded that two factors are necessary for the production ofthe abnormal electrocardiogram, lesions of the conduction systemand cardiac fatigue. Wilson and Herrmann, in a recent publica-tion, state that complete bundle b
Clinical electrocardiography . Fig. 84.—Right bundle branch Fig. 85.—Left bundle branch block. subendocardial lesions were produced without the developmentof Q R S changes. Abnormal complexes were obtained, however, 106 CLINICAL ELECTROCARDIOGRAPHY by dividing the bundle branches, and the complexes becamedefinitely bizarre when ventricular dilatation supervened. Smithconcluded that two factors are necessary for the production ofthe abnormal electrocardiogram, lesions of the conduction systemand cardiac fatigue. Wilson and Herrmann, in a recent publica-tion, state that complete bundle branch block is capable of pro-ducing characteristic Q R S changes, but they do not believe thatlesions of the bundle subdivisions are capable of producing suchchanges. Notwithstanding the variance of views with regard to themode of production of the abnormal QRS complexes, these electro-cardiograms, when all derivations are involved, are indisputablylinked with a clear-cut clinical picture. Patients having this dis-order have marked myocardial disintegration, a
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