Pulmonary consumption, pneumonia, and allied diseases of the lungs : their etiology, pathology and treatment, with a chapter on physical diagnosis . the inner and outer fibrous coats suffer from markedcellular infiltration, and the cartilages and layerweaken and become atrophied. The muscular coat of thesmall arteries is usually thickened, but the Avails of thecapillaries are thin and distended into large sinuses, the air-vesicles, as a result of the forcible coughing efforts incidentalto chronic bronchitis, are overdistended, which leads topermanent emphysema. Fig. 44
Pulmonary consumption, pneumonia, and allied diseases of the lungs : their etiology, pathology and treatment, with a chapter on physical diagnosis . the inner and outer fibrous coats suffer from markedcellular infiltration, and the cartilages and layerweaken and become atrophied. The muscular coat of thesmall arteries is usually thickened, but the Avails of thecapillaries are thin and distended into large sinuses, the air-vesicles, as a result of the forcible coughing efforts incidentalto chronic bronchitis, are overdistended, which leads topermanent emphysema. Fig. 44 describes the pathologiccondition of the tube. * For a description of the •normal histology of the bronchi thereader will refer to p. 252. CHRONIC BRONCHITIS. 493 It will be seen that the whole bronchial wall is thickened,thrown into folds, and is densely infiltrated. The basementmembrane is easily made out, and on it are small bud-likeprojections, germinating epithelial cells. The air-vesiclesaround it are markedly emphysematous. 2. When Dependent on Valvular Lesions of the lesions of the heart always interfere with the free.
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