Lectures on nervous diseases from the standpoint of cerebral and spinal localization, and the later methods employed in the diagnosis and treatment of these affections . e indispensablein deciding as to the existence of softening, if in the anterior part of the 318 LECTURES ON NERVOUS DISEASES. frontal, the occipital, or the temporal lobes, where the so-called motorcentres of the brain are wanting. (See Fig. 5.) There is more or less oedema into the brain-substance which sur-rounds a spot of red softening. This often causes the area of softeningto appear as an elevation abine the plane of the
Lectures on nervous diseases from the standpoint of cerebral and spinal localization, and the later methods employed in the diagnosis and treatment of these affections . e indispensablein deciding as to the existence of softening, if in the anterior part of the 318 LECTURES ON NERVOUS DISEASES. frontal, the occipital, or the temporal lobes, where the so-called motorcentres of the brain are wanting. (See Fig. 5.) There is more or less oedema into the brain-substance which sur-rounds a spot of red softening. This often causes the area of softeningto appear as an elevation abine the plane of the section when the brain-tissue is divided with the knife. In cases where the focus of softeningis of the white or yellow variety tliis elevation is wanting. The debris which constitutes a focus of cerebral softening will befound to consist of fat granules, altered blood-corpuscles, pigmentmasses, pus-cells, disintegrated nerve-tissue, caseous matter, and largespherical cells, filled with fat granules (Gluges corpuscles). Eachvariety of softening causes variations in the relative proportions of theseelements. The red variety will exhibit a large admixture of blood-. FiG. So —Softened Brain-Tissue (After Fox ) corpuscles, pigment granules, and Gluges cells; the yellow and whitevarieties will be accompanied b} an excess of fatty matter or caseoussubstance, and a small admixture of altered blood pigments, corporaamylacea, and crystals. Etiology.—It is a mistake to suppose that cerebral softening occursonly in the aged. Old age is indeed a preeminent factor in exciting cere-bral thrombosis; and that condition is followed, as a rule, b} softening ofthe brain in the old. Still, the more common exciting causes of this dis-ease include (in addition to cerebral enil)olisin and thrombosis) pyjemia,syphilis, alcoholism, sun-stroke, and cereln-al hemorrhage, all of whichmay exist independently of old age. I have to-day under my care ayoung married lady (thirty years old) who has marked c
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