. A treatise on the nervous diseases of children, for physicians and students. apter, and it did not seemdesirable to add them to the sections on congenital nuclear palsies. MENINGITIS. 419 of progressive bulbar palsy. This disease in the adult bears a close clinicaland anatomical relation to progressive amyotrophy and to amyotrophic lateralsclerosis. With the chronic diseases of adults we are not concerned, but ashort allusion may be made to progressive bulbar palsy of earlier years,which is a rare disease, but, like the adult form, is due to a primary degen-eration of the nuclei of the lower
. A treatise on the nervous diseases of children, for physicians and students. apter, and it did not seemdesirable to add them to the sections on congenital nuclear palsies. MENINGITIS. 419 of progressive bulbar palsy. This disease in the adult bears a close clinicaland anatomical relation to progressive amyotrophy and to amyotrophic lateralsclerosis. With the chronic diseases of adults we are not concerned, but ashort allusion may be made to progressive bulbar palsy of earlier years,which is a rare disease, but, like the adult form, is due to a primary degen-eration of the nuclei of the lower cranial nerves.* A case of this descrip-tion, from my clinic, was carefully studied and described by Dr. Wiener. The symptoms of unilateral bulbar palsy were discovered quite acciden-tally in a young man who had been under treatment by his physician for tu-bercular glands and pharyngitis. He presented : 1. A very marked deviationof the tongue to the right when protruded. 2. Distinct atrophy of the me-dian portion of the right half of the tongue. 3. The faradaic response of. Fig. 104.—Same Case as Fig. 103. Hypoglossal nucleus, right side, showing absenceand extreme degeneration of ganglion cells. the right half of tongue was much diminished ; the contractions were slug-gish, there was increased galvanic excitability of the right side (K. C. ;A. C. C.); the contractions were slow and wave-like. 4. Taste and tactile sen-sations were normal. 5. Deviation of the soft palate and of the uvula to theleft side. On phonation the paralysis of the right side became more Great difficulty in deglutition. 7. Dysphonia due to disease of the rightrecurrent laryngeal nerve. Other cranial nerve functions were normal. All reflexes normal. A gradual progression of all the symptoms led to a fatal issue from respira-tory failure. Examination of the brain revealed a distinct degeneration of thehypoglossal and vago-accessorius nuclei of the right side, with degenerationof the respira
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