. The effects of inanition and malnutrition upon growth and structure . Fig. 45.—Chart showing growth curves of young rats fed on diet deficient in both vitamins A and B. The curves represented by dots (....) show the inability of the rats to grow when both vitamins, A and B, are absent from the diet. When A alone is added, , in butter fat, (curves marked ), there is no improvement; but upon adding both A and B (curves marked ), excellent growth follows. When vitamin B alone is added, there is sometimes slight growth (curves marked xxxx), probably due to unexhausted reserve stores of A, bu
. The effects of inanition and malnutrition upon growth and structure . Fig. 45.—Chart showing growth curves of young rats fed on diet deficient in both vitamins A and B. The curves represented by dots (....) show the inability of the rats to grow when both vitamins, A and B, are absent from the diet. When A alone is added, , in butter fat, (curves marked ), there is no improvement; but upon adding both A and B (curves marked ), excellent growth follows. When vitamin B alone is added, there is sometimes slight growth (curves marked xxxx), probably due to unexhausted reserve stores of A, but growth failure eventually follows. (Drummond; Med. Res. Comm. '19.) apparent effect on the offspring. When such diets are fed to the pregnant female, still-births are more frequent. Even though the newborn may appear normal, they are unusually susceptible to rachitoid disorders later, especially when the deficient diet of the mother is continued during lactation. The relation of vitamin A to the cause of rickets was mentioned above in connection with mineral deficiencies. The characteristic ocular lesions (xeroph- thalmia) produced by deficiency of vitamin A will be discussed in Chapter XIII. Vitamin B. Polyneuritis and Beriberi.—The importance of vitamin B in the production of polyneuritis in animals (especially birds) and of human beriberi became evident through the work of Eijkman ('97) and numerous later investi- gators. For review of various other theories of beriberi, see Vedder ('13, '23) and Vaughan ('23). Nagayo ('23) claims that human beriberi, although it may involve a deficiency of vitamin B as one factor, is a disorder distinct from experimental polyneuritis and more closely resembles the infantile "Mehlnahr- ; The marked atrophy of the body in experimental polyneuritis does not occur in human beriberi. The failure of growth in young animals on diets deficient in vitamin B was demonstrated by Funk and Macallum ('15), Osborne and Mendel ('17a
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