Manual of pathology : including bacteriology, the technic of postmortems, and methods of pathologic research . ory and necroticprocesses are |)artly enca])sulated. Anemia, hyperemia, and congestion of boneare of little pathologic importance when con-sidered as simple processes. The absence ofelasticity prevents any unusual degree of vas-cular distention, and therefore limits the possi-bility of abundant arterial influx or venousaccumulation. This statement does notimply that the amount of blood within the p,^ jpc^-cuROMr rRn„tcT,vEbone can not vary. Variations, however. Ostko periostitis. are
Manual of pathology : including bacteriology, the technic of postmortems, and methods of pathologic research . ory and necroticprocesses are |)artly enca])sulated. Anemia, hyperemia, and congestion of boneare of little pathologic importance when con-sidered as simple processes. The absence ofelasticity prevents any unusual degree of vas-cular distention, and therefore limits the possi-bility of abundant arterial influx or venousaccumulation. This statement does notimply that the amount of blood within the p,^ jpc^-cuROMr rRn„tcT,vEbone can not vary. Variations, however. Ostko periostitis. are slight, and even with extensive inflam-matory processes the evidence of distention may be general anemia, and when obliterative lesions in the blood-vessels,such as result from syphilis, have affected the arteries leading tothe bone, a varving amount of anemia may occasionally be recog-nized. During the active stage of osseous evolution and growth, andbefore the completion of development, the blood-supply to the bonesis relativelv abundant. With completion of the osseous matrix, and. 8o6 SPECIAL PATHOLOGY. later, when marrow transformation has been accomphshed, the amountof blood present in the bone is greatly diminished. During variousprocesses the blood-supply is manifestly increased, and retardation ofexit may intensify vascular distention, but the inelasticity of the Haver-sian canals more or less limits even inflammatory hyperemia. Thechanges in the bone-marrow seen in pernicious anemia have been regardedby some authorities as evidence of hyperemia. The conversion of thefatty marrow into the richly cellular marrow, as is sometimes seen inthat disease, as well as in leukemia, certainly implies an increased blood-supply ; but here the increased supply of blood constitutes a part of thelocal formation of new marrow, and is scarcely to be considered, in thepresent state of our knowledge, as a primary medullary process. Hypertrophy of Bone.—In giants th
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