Manual of pathological anatomy . utaneous tissue of the neck, thorax, and body is well known that a similar condition of the subcutaneous tissuemay arise from laceration of the trachea. These occurrences arecomparatively rare, and not imjDortant. Development of gas inthe body from putrefaction after death is sometimes called post-mortem emphysema, a condition only mentioned to prevent itsbeing confounded with that of which we have now to speak. VESICULAR EMPHYSEMA. Dr. Baillie, though unable to suggest the means of distinguishingthis disease before death, was one of the first to s
Manual of pathological anatomy . utaneous tissue of the neck, thorax, and body is well known that a similar condition of the subcutaneous tissuemay arise from laceration of the trachea. These occurrences arecomparatively rare, and not imjDortant. Development of gas inthe body from putrefaction after death is sometimes called post-mortem emphysema, a condition only mentioned to prevent itsbeing confounded with that of which we have now to speak. VESICULAR EMPHYSEMA. Dr. Baillie, though unable to suggest the means of distinguishingthis disease before death, was one of the first to show its true naturein the dead body. It consists essentially in a dilatation of a largeror smaller number of air vesicles, and may be produced by anycause exerting a great strain upon them. The effect is to diminishthe specific gravity of the part affected, so as to render it morebuoyant than the healthy lung tissue in water; to cause the lungto become less crepitant on compression, giving it a doughy or 460 VESICULAR Portion of emphysematous lung—the cavities areeither formed by simply enlarged air cells, or by thecoalescence of groups of cells. (St. Bartholomews Museum, xiv. 11) woolly feel, to prevent its collapse on tlie thorax being opened, andto render it more or less dry and exsanguine. The emphysematous portion, if superficial,Fig. 102. projects above the sur- face of the unaffected13art; and large bullaemay be visible on thesurface of the lung,from the gradual oblite-ration of the intervesi-cular tissue, allowingseveral vesicles to loss of elasticity inthe pulmonary tissue,whether primary or se-condary, prevents theusual collapse of thelung at the period ofexpiration; the vitiatedair is not expelled as itought to be; and this isone cause of want ofoxygen. Another cause of deficient respiration is the diminution ofthe respiratory surface. As the grouj^s of air cells expand and theirpartitions become obliterated, and still more when the walls arer
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