General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . Fig. 3f>7.—A chronic alveolar abscess(dental granuloma) in the lower rightcuspid, right central incisor, left centralincisor and left cuspid. The apices ofthe roots of the central incisors have un-dergone marked resorption. osseous tissue in chronic dentoalveolar abscess typifies tissuedeath by consecutive disintegration of cellular elements throughrarefying osteitis, osteoporosis or caries, practically synony-mous terms; or in other words, gradual cell death,
General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . Fig. 3f>7.—A chronic alveolar abscess(dental granuloma) in the lower rightcuspid, right central incisor, left centralincisor and left cuspid. The apices ofthe roots of the central incisors have un-dergone marked resorption. osseous tissue in chronic dentoalveolar abscess typifies tissuedeath by consecutive disintegration of cellular elements throughrarefying osteitis, osteoporosis or caries, practically synony-mous terms; or in other words, gradual cell death, as already 4GG DENTAL PATHOLOGY described in the case of osteomyelitis leading to rarefying ostei-tis, or to the more complete process of hard-tissue resorption withliquefaction of the organic hone matrix—caries of bone (). Necrosis of areas of alveolar process and jaw bone properis occasionally observed in connection with ulcerative periosteum at the crest, or alveolar border, becomes involved,. Fig. 368.—Chronic dentoalveolar abscess (dental granuloma) of very long standing inwhich the chronic osteomyelitis which developed in the alveolar process resulted incaries of hone in a relatively large area. The apex of the tooth was found to penetrateinto a cavity in the cancellated substance of the maxilla. the infection brings about its detachment, and the involvementof the myeloid substance in the cancellated spaces follows. Theblood supply is cut off from the periosteal side and infectiousemboli obstruct any number of capillaries in the cancellatedsubstance, with necrosis as the result. CHAPTER XXXVI PERIOSTITIS OF THE JAW Inflammation of the periosteum of the jaw may be acute orchronic, depending on the vital resistance of the patient and thevirulence of the infecting organism. The mosl frequent causes of periostitis of the jaws are: 1. Acute or chronic dentoalveolar abscess. 2. Traumatic injuries with or without a communicating ex-ternal wound. 3. Ch
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Keywords: ., bookcentury1900, bookdecade1920, booksubjectpathology, bookyear19
