General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . the investing tissues of salivary de-posits and of subgingival deposits as originally pointed out byG. V. Black. In the former, well-defined pus pockets are rare,while in the latter form pockets almost invariably follow. Theearly deposition of subgingival calculi can be detected by thecolor of the gingivae, which are of a deep red, in some instancesrunning to a blue, purple, or even black. The tissues are flabbyand present an appearance typical of a chronic inflam
General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . the investing tissues of salivary de-posits and of subgingival deposits as originally pointed out byG. V. Black. In the former, well-defined pus pockets are rare,while in the latter form pockets almost invariably follow. Theearly deposition of subgingival calculi can be detected by thecolor of the gingivae, which are of a deep red, in some instancesrunning to a blue, purple, or even black. The tissues are flabbyand present an appearance typical of a chronic stratified squamous epithelium dips into the connective tis- SI BGINGIVAL DEPOSH - :;:»:; sue to a greater Length than is the case with normal epithelium,and the connective-tissue mat of the mucous membrane is almosteverywhere infiltrated with Large masses of round cells, most ofwhich are mononuclear Leucocytes, large and small, plasma cells,lymphocytes and polymorphonuclear leucocytes in small numbers(Figs. 317, 318 and 319). Mast cells so often found inmucous membranes which are the seat of chronic inflammation. Fig. 317.—Section of human gingiva. The stratified squamous epithelium has lostits characteristic appearance; the projections of epithelial cells between the papillae ofconnective tissue have become elongated. The gingiva was the seat of a chronic inflam-mation which by continuity involves the peridental membrane and the alveolar marks the beginning of pocket formation. a, a, elongated epithelial projections; b,connective-tissue stroma. are also to be seen. The infection spreads slowly as a generalrule, and. involving the fibers of the peridental membrane and thealveolar process, initiates the progressive process of destructionin the supporting tissues of the tooth (Figs. 320-325). The pus 594 DENTAL PATHOLOGY pocket is the result of the destruction of the peridental fibersand of the alveolar process. The proteolytic toxins formed inthe course
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Keywords: ., bookcentury1900, bookdecade1920, booksubjectpathology, bookyear19
