Nervous and mental diseases . Fig. 235.—Sitting attitude in paralysisagitans. Fig. 236.—Parkinsons disease : facies. creased fades. % tremble. Owing to the rigidity, this reaction-time is increased about forty per cent. Passive motion is sometimes impeded by the rigidity, but there is no hypertonus and the tendon reflexes are only slightly in- A foot-clonus is never encountered. There is a characteristic The nasolabial folds and lines of expression tend to disappear. The face becomes smooth. The brow may retain its cross-wrinkles through enforced V |^^ elevation requisite for forward vision, i
Nervous and mental diseases . Fig. 235.—Sitting attitude in paralysisagitans. Fig. 236.—Parkinsons disease : facies. creased fades. % tremble. Owing to the rigidity, this reaction-time is increased about forty per cent. Passive motion is sometimes impeded by the rigidity, but there is no hypertonus and the tendon reflexes are only slightly in- A foot-clonus is never encountered. There is a characteristic The nasolabial folds and lines of expression tend to disappear. The face becomes smooth. The brow may retain its cross-wrinkles through enforced V |^^ elevation requisite for forward vision, if ^ n| tne body and bead be bowed. The eyes \V are widely opened and rarely wink. The V 9p ocular globes tend to remain fixed so that \^\?* the patient, in order to change the direction of vision, ordinarily turns bodily with the *jv neck held rigid. Movements of ocular -^^J g* association and accommodation are im- ^^m peded by this rigidity, and the patient ?HMMtfr- —r^1 H shows little or no facial variation for any
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