The Journal of laboratory and clinical medicine . d tissue eitherbeing destroyed by the pressure of the surrounding connective tissue, or pro-liferating and adenomatous acini in small encapsulated areas like those foundin old degenerating goitres. These follicles contain no colloid but are glandacini with columnar or high cubical epithelium; only very rarely is seen anatrophic follicle with a small mass of shriveled colloid. There is one area ofthis adenomatous tissue slightly larger than the rest and well encapsulated. Theamount of glandular tissue as a whole is extremely small. The picture p
The Journal of laboratory and clinical medicine . d tissue eitherbeing destroyed by the pressure of the surrounding connective tissue, or pro-liferating and adenomatous acini in small encapsulated areas like those foundin old degenerating goitres. These follicles contain no colloid but are glandacini with columnar or high cubical epithelium; only very rarely is seen anatrophic follicle with a small mass of shriveled colloid. There is one area ofthis adenomatous tissue slightly larger than the rest and well encapsulated. Theamount of glandular tissue as a whole is extremely small. The picture presentedby this thyroid is unique in that it is a true chronic interstitial productive thy-roiditis. There is marked increase of connective tissue, old connective tissueshowing hyaline change, and areas of young connective tissue around areas ofextreme round-celled infiltration. This inflammatory reaction has gone to thepoint of alnidst total glandular destruction, so that only small islands of gland- General Pathology of Pellagra 225 PLATE III,. f^ ri-. •^ „^ ... >J; •; w .tf >^ #?.?v--V^ Fig. 7. Adrenal, low power, showing dilated bloodspaces and fibrosis of medulla.
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Keywords: ., bookcentury1900, bookdecade1910, booksubject, booksubjectmedicine