General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . Fig. 378.—Chronic gingivitis in the gingiva which by process of continuity will spri adto tin d peridental membrane, a, a cementum; b, b, dentin; c,c,c, stratified ous epithelium lining gingival cul-de-sac; d, large area of round cell infiltra(predominance of mononuclear wandering cells). \\ ORRHEA \1.\ EOLARIS 479 find lodgmenl between the teeth in the presence of either defective approximal contacts (Figs. 371, 372, 373, 374, and 375^, orin tlio absence of conta
General and dental pathology with special reference to etiology and pathologic anatomy; a treatise for students and practitioners . Fig. 378.—Chronic gingivitis in the gingiva which by process of continuity will spri adto tin d peridental membrane, a, a cementum; b, b, dentin; c,c,c, stratified ous epithelium lining gingival cul-de-sac; d, large area of round cell infiltra(predominance of mononuclear wandering cells). \\ ORRHEA \1.\ EOLARIS 479 find lodgmenl between the teeth in the presence of either defective approximal contacts (Figs. 371, 372, 373, 374, and 375^, orin tlio absence of contacts; by tiegled of the teeth through in-sufficienl brushing, etc.; by the rough edges of fillings or crownbands; by salivary calculi; by severe manipulations of ligaturesand rubber dam clamps; or by improper handling of the tooth-brush, producing injuries of the gingival margins, an inflamma-tory process is inaugurated in these tissues (the Eree gingiva?) ac-companied by the depositions of calcareous masses Fig. 379. Decalcified transverse section of upper central incisor. In the gum tissue;i chronic inflammatorj process is going on which has resulted in ana- of tissue liquefac-tion. In this instance the chronic gingivitis had not as yet involved the alveolar process,although eventually it will il so. alveolar process; b, b, areas of tissue liquefactionin stroma of mucous mfembrane of gum tissue; c, cementum; d, dentin; e, pulp. The subgingival deposits thus produced irritate and subsequentlybring about an infectious inflammation of the gingivae, gums, andlater of the peridental membrane immediately adjacent to them(Figs. 376-382). Through the action of Liquefying bacterial tox-ines the peridental libers disappear and the infectious inflamma-tion, as soon as the cresl of the alveolar process is reached, entersthe bone and there sets up an osteomyelitis with the absorptionof the bony Lamella (Fig. 383). The inflammation extends again, 480 DENTAL PATHOLOGY
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Keywords: ., bookcentury1900, bookdecade1920, booksubjectpathology, bookyear19
